A groundbreaking new study led by researchers at the Masonic Medical Research Institute (MMRI) has identified a promising molecular target to protect the heart against damage caused by high-fat diet and obesity. The study, published this week in the leading journal Science Signaling, highlights the protein tyrosine phosphatase PTP1B, a nodal enzyme involved in insulin signaling, as a key driver in maladaptive cardiac metabolism and dysfunction under dietary stress.
In obesity and high-fat diet conditions, the heart undergoes a metabolic switch, moving from its healthy preference for fatty acid oxidation to an overreliance on glucose. This shift contributes to cardiac steatosis, mitochondrial dysfunction, and ultimately a cardiac…
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